Journal of Virology
- Amino acid substitutions in NSP6 and NSP13 of SARS-CoV-2 contribute to superior virus growth at low temperatures
- Plus-strand RNA viruses hijack Musashi homolog 1 to shield viral RNA from cytoplasmic ribonuclease degradation
- The antiviral BDGR-49 provides protection from lethal, neurotropic Venezuelan equine encephalitis virus intranasal infection in mice
- FBXO45 restricts HIV-1 replication by inducing SQSTM1/p62-mediated autophagic degradation of Tat
- Evolution of ubiquitin, cytoskeleton, and vesicular trafficking machinery in giant viruses
- Diversities of African swine fever virus host-virus dynamics revealed by single-cell profiling
- Differential mosaicism of recombinant foot-and-mouth disease viruses resulting from heterologous superinfection of cattle
- Begomovirus capsid proteins interact with cyclic adenosine monophosphate (cAMP)-specific phosphodiesterase of its whitefly vector and modulate virus retention within its vector
- Activation of CXCR3+ Tfh cells and B cells in lymph nodes during acute HIV-1 infection correlates with HIV-specific antibody development
- Seneca Valley virus 3C protease cleaves HDAC4 to antagonize type I interferon signaling
- POLM inhibits porcine epidemic diarrhea virus replication by degrading multiple viral structural proteins
- A conserved lysine/arginine-rich motif is essential for the autophagic degradation of potyviral 6K1 protein and virus infection
- Generation of RNA aptamers against chikungunya virus E2 envelope protein
- IFI16 recruits HDAC1 and HDAC2 to deacetylate the Kaposi’s sarcoma-associated herpesvirus (KSHV) latency-associated nuclear antigen (LANA), facilitating latency
- Interactions between human immunodeficiency virus and human endogenous retroviruses
- Distinct mutations emerge in the genome of serotype O foot-and-mouth disease virus during persistence in cattle
- Longitudinal analysis of rhesus macaque metabolome during acute SIV infection reveals disruption in broad metabolite classes
- Contrasting roles of PSGL-1 and PD-1 in regulating T-cell exhaustion and function during chronic viral infection
- Seneca Valley virus induces mitochondrial apoptosis by activating ER stress or the PERK pathway based on Ca2+ transfer from ER to mitochondria
- Conformational activation and disulfide exchange in HIV-1 Env induce cell-free lytic/fusogenic transformation and enhance infection